Tate Tabtieng

Tate

Tate Tabtieng
BE, Bioengineering
University of Pennsylvania
Philadelphia, PA
Biochemistry PhD Student
Marta Gaglia, PhD, Adviser

 

 

 

Kaposi’s sarcoma-associated herpesvirus (KSHV) is a herpesvirus that infects B lymphocytes and endothelial cells, and causes tumors in immunocompromised patients. Like other herpesviruses, KSHV undergoes cycles of latent and lytic infection. Reactivation of KSHV into the lytic cycle is an essential process for tumor formation. To accomplish lytic reactivation, KSHV must avoid innate cellular immune responses. My research focuses on the role of caspases in circumventing the antiviral immune response and promote lytic reactivation. Understanding the mechanisms behind lytic reactivation of KSHV can provide key insights for therapies to block tumor development.

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February 15: Building Diversity in Biomedical Sciences

March 31: Post-Baccalaureate Research Program

May 1: Clinical & Translational Science, MS in Pharmacology & Drug Development

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